Page 2 of 2

FEMALE HORMONES

Abstract

Did you know that it’s best to start treatment with estrogen BEFORE 60? The study that scared everybody from taking estrogen 25 years ago had many flaws. I will review them. It’s estrogene in conjunction with synthetic progesterone, gestagen, that can lead to an increased risk for breast cancer and heart issues. And especially if starting after 60. It’s also very important what kind of estrogen you take. Estradiol, which is plantbased, is better for us than conjugated estrogens. Premarin. Premarin is made from the urine from pregnant mares. The mares are being abused in the process; horrible to read about. And transdermal is better than oral. 

Nb! When you take ORAL estrogen, the total hormones become higher. The free hormones must be taken. 

Natural progesterone is also better than synthetic. It’s also plant based, derived from the same plant as oestradiol, wild yam.  It’s only the natural that is called progesterone, synthetic is called things like gestagen and progestins.

If you still have your uterus, it’s very important to take progesterone when you use estrogen. If not, the uterine wall might become thicker. It seems it’s mostly natural progesterone in pillform that has this effect on the uterine wall. Though my progesterone cream kept my uterine wall normal.

One  does not find any increased risk of breast cancer when estrogen is combined with natural progesterone, only when combined with synthetic.  Natural progesterone also has a beneficial influence on the cardiovascular system. 

Testosterone is also very important for us women, not only for men. It’s important for sexual excitement, the ability to orgasm, muscle strength and our bones. And the ability to hold our water! Anyone having issues with that?😜

Continue reading

FOR THE THYROID NEWBIE

Basic guide for you who are new to thyroid issues

I wanted to make the guide I wish I had had. Way back when, more than 20 years ago. Knowledge that could have saved me a lot of suffering. Maybe I would have been more healthy today.

So it is my hope, this info will help you. Of course, there is so much info today, compared to earlier. But maybe there is too much info for someone new to this. I hope I can make it,if not easy, then at least possible to understand.

I don’t know how you are feeling at present. If you have just been diagnosed with hypothyroid, with autoimmune thyreoditis, have had your thyroid removed or maybe you are struggling to get a diagnosis. Maybe your doctor is dismissing you, but you feel sick and know something is wrong.

Continue reading

ESTROGENS IN MENOPAUSE, time to change the recommendations?

Translation from an article by E.F.Eriksen, M.H.Moen, O.L. Iversen,

This is a translation of an article published in the Norwegian Journal for Physicians, Tidsskriftet, https://tidsskriftet.no/2018/03/kronikk/ostrogener-i-menopausen-p%C3%A5-tide-endre-anbefalingene

New studies show, that  treatment with estrogen with start before age 60, are for the most part positive.  It’s time to change the recommendations.

Continue reading

REVERSE T3

Abstract

Reverse T3 is not the big bad wolf in our thyroid system. It is a necessary brake where FT3 is the gas pedal. We can get too much RT3 when we take more T4 medicine than we are able to convert to T3. Could be just too high dose, or that we don’t convert well. Other conditions can increase RT3 too much as well. It’s a good idea to test RT3 from time to time. But it is not necessary to test all the time. 

I will look at myths and misunderstandings around RT3. for one thing, RT3 does not block T3 by occupying T3 receptors. Nor does it hinder T4 to T3 conversion. If there is a big bad wolf, it’s deiodinase 3, D3. It’s D3 who converts T4 to RT3, and who hinders T3 from attaching to the T3 receptors i the cell nucleus. 

But there are no “wolves” in our thyroid system. Only participants with their own roles to play. And the better we understand the play, the more can we hopefully optimize our thyroid levels and feel better. 

I will look at what causes too high RT3, and what one can do bring it down. 

Continue reading

THE IODINE PROTOCOL

As with LDN, I am no expert on Iodine. I am on it myself, have been for about 6 months here in may 2020. I will be updating the post as I go along. I will be absolutely honest about what is happening. I won’t be writing too much about the complicated aspects of Iodine. But I want to present some of it. So if you are mostly interested in the more practical aspects and concrete tips, scroll down to “The Protocol”.

Continue reading

HAS NDT GONE BAD?

NO, NDT hasn’t gone bad. That is the short answer. I take NDT myself, and it works just fine.

I see people claiming, NDT is no good anymore. That it is better to take synthetic T4/T3 now. I think this is very serious, that this incorrect, untrue info is being spread in the Facebook groups. It stems from STTM, Stop the Thyroid Madness, and its Facebook group, Adrenal Fatigue and Thyroid Care. STTM is a site that has contributed so much, and I am sad to see, there is such black and white thinking going on. This issue isn’t black and white.

I wrote to the woman behind STTM about this, Janie A. Bowthrope. I told her, we are lots of people who do great on NDT here in Scandinavia. She said, most of those who still “believe”  they are ok on NDT, only experiences adrenalin! I find that to be a strange claim. What does it mean? Adrenalin? We don’t have good thyroidlevels? We just think we do? A female admin in the Facebook group, told me in a comment, we here in Scandinavia probably took our meds right before labs! None of us know how to do our labs (?) I am a member in thyroidgroups  in many countries. The members in the Norwegian groups are among the better informed in the world, as far as I am concerned. I am aware that many thyroid patients don’t know how to do labs correctly. Norwegians on NDT are not among those.

It’s a little difficult to take this seriously. Yes, absolutly, there were some bad batches of Nature Troid in 18. It didn’t take long before there were good batches again. STTM claims, that NP Acella is bad now. We don’t have NP here in Scandinavia, I don’t know if they have it in other European countries. But i see lots of people saying it is fine for them, in the US groups. Just now, May 2020, I see people here in Scandinavia saying, there is something wrong with Erfa. With the smelly product Erfa themselves have made a statement about it. Erfa says, they contain the proper amount of hormone. But some experience it differently.  

So I am not saying, there are no issues. But to say, all NDT has gone bad, and to accuse the producers of being callous and indifferent to us patients and only out to make money, as Janie Bowthrope does here, https://stopthethyroidmadness.com/2019/09/23/the-sad-saga-of-where-we-are-today-as-hypothyroid-patients/  That is a sad saga.

Continue reading

SYNTHETIC T4 TO NDT CONVERSION

How do I convert my T4 dose to NDT?

This is a question one sees often in the thyroid Facebook groups. There is a lot of confusion and insecurity around this. Which is not so strange, as there is very little good info on the topic. Here are my recommendations

The conversion tables the NDT (thyroid) producers themselves provide are ridiculously low. They are wrong, wrong, wrong. I don’t understand how they think at all. If they do. Think. One would be seriously under dosed if adhering to those guidelines. And if one’s doctor reads them, one could get serious problems. I don’t know how many doctors read these instructions. I hope they don’t. Here is a table from Nature Troid as a (bad) example.  

Continue reading

CONVERSION, DEODINASE

I have translated some of Thyroid Patients Canada’ articles on conversion into Norwegian. These articles are very interesting, esp this one, https://thyroidpatients.ca/2019/11/12/the-basics-of-thyroid-hormone-action-transport-and-conversion/      

Most of the hormone conversion, T4 to T3 happens outside the thyroid. though there are big individual differences how much.  There are 3 enzymes responsible for this conversion, D1, D2 and D3. They do a lot more than convert T4 to T3 though.

Continue reading

FT3 UPS AND DOWNS IN T3 THERAPY

This is not really a post. I have translated Tania Sona Smith’s article, https://thyroidpatients.ca/2019/11/25/free-t3-peaks-and-valleys-in-t3-and-ndt-therapy/ , into Norwegian.  For every Norwegian page, I want there to be a corresponding English, and vice versa.  

This is just a presentation of Tania’s piece. She is looking into the reseach on how Ft3 behaves after dosing with T3 containing medicine.  Actually, there is not really any good studies on the subject . There is a study on FT3 after a single dose of T3. But that is not what most people take when dosing with T3. We do multidoses. There is no study on FT3 when multidosing, but there is one on T3, that is total T3. Where I live in Denmark, we cannot have FT3 tested. The endoes have forbidden it, they claim, FT3 fluctuates too much, it cannot be trusted. How do they know? With no studies? Anyway, FT3 and T3 follow each other in most instances . With a very important exception, when we take estrogen orally. Then both T4 and T3 becomes higher and FT4 and FT3 lower. So  VERY important to have the frees tested.

Anyway, there is a study on multidosing and T3, showing that T3 peaks app 2 hours after intake. So pretty unstable. Except of course, the peaking is very easy to predict. At least for us patients. I don’t know if the Danish endos,( and the English) have heard about the single dose experiment (Jonklaas et al, 2015) maybe, where one sees a huge peak after intake of 50 mcg T3 in one go. If that is the fluctuation that has scared the shit out of them. And then they never got around to reading  Busnardo et al’s study(1980) on total T3 and multidosing. Even though that study has been around for 40 years. 

Tania goes into all this and more.  We really need a good study on the FT3 level through the day and night. Preferably for all three T3 containing meds, Mono T3, synthethic T4/T3 and NDT.  And with differing dosingregimes. We are many today who also take a dose of T3 at bedtime, others set the alarm 2 hours before getting up, to provide T3 for the cortisolproduction that takes place in the early morning.  Considering how FT3 probably peaks 2 hours afterdose, that is maybe not the best time to take it. Anyway, such a study would be great.

TRIAC

This is just a short abstract of Thyroid Patients Canada’s article on Triac, https://thyroidpatients.ca/2020/01/02/when-dosing-t3-you-get-higher-levels-of-triac/ . I just want to draw your attention to this metabolite. The quistionmark on the image, is meant to signify my surprice when learning of Triac. I had never heard of this thyroidhormone metabolite until I read Tania’s post. It is quite strange, as it has been studied since the 1950ies. I don’t think many of our doctors have ever heard of it.

So what is it? It is a thyroidhormone metabolite like FT4 and FT3 are. It acts much as FT3, it’s abbreveration is TA3. It has a much shorter halflife than FT3, about 6 times faster.

Continue reading

Newer posts »

© 2021 THYROIDBLOG.COM

Theme by Anders NorénUp ↑

Good news about a new T3 medication!

There seems to be a new T3 medicine ahead, PZL. It has a slower uptake and a smaller FT3 peak. As well as a longer half life.  You can read more about it